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CyrusManz

CyrusManz
Joined Jan 2022
Bio

61 year old active male, clinically diagnosed with Central Sleep Apnea in 2020.

CyrusManz
Joined Jan 2022
Bio

61 year old active male, clinically diagnosed with Central Sleep Apnea in 2020.

Your Oscar chart shows an incredibly high number of CA events, highly unusual. A BiPap machine algorithm basically looks at the flow rate and if your airway is open but you are not exhaling, then there is no resistance on the flow rate and your machine algo registers this event (if it lasts more than 10 seconds) as a "central" event. I am guessing that you have been given a Resmed machine but without its model, I can't be much help on correcting the titration in order to get rid of the centrals because it could very well be that you are not having that many central events and the machine algo is incorrectly reporting it because of titration. Also, what was the result of your original sleep study? (was it 90% CSA and 10%OSA for example?) I am asking because your doctor should have prescribed a ASV machine instead of a bipap if your CSA events were higher than 20% of the time during your sleep study. The reason you need a ASV type is because the algorithm on an ASV is designed to monitor and react to your respiratory function, on a breath-by-breath basis, so it will constantly adjust its IPAP and EPAP pressures within a wide range in order to "stabilize" your "Minute Ventilation".

A non-ASV BiPAP is only designed to drop EPAP pressures to a preset limit at expiration and vice versa for inhalation. The algorithm does not otherwise attempt to stabilize Minute Ventilation or any other respiratory characteristics. It just provides positive pressure to keep your upper airway open in order to treat your OSA with the added comfort and precision of having a secondary pressure setting for exhalation.

Look up Adaptive Servo Ventilation (ASV) therapy online. There are a lot of very good articles about it. The machine itself looks exactly like a CPAP machine but the turbine and software are designed for ASV function.

Well there is a clear scientific observation that connects patients with CHF to CSA. So they believe that CHF can lead to CSA and vice versa. Some scientists want to start studies into whether or not the development of CSA is a natural response to CHF in order for the body to moderate its PaCO2 levels...THAT is way out in the woods for me:-)

Studies also show that "treatment emergent CSA" is a thing, meaning that the long term CPAP therapy for OSA, leads to the development of CSA. (Remember that CSA itself was only isolated as a disorder back in 2008 at Mayo clinic so it is a relatively new subject and under intense study, internationally.) BUT I have also seen studies that suggest " treatment emergent CSA" will go away by itself within 6-8 weeks, after OSA CPAP therapy stops, but who would stop their therapy, unless they get an "implant surgery", like "INSPIRE" at a cost of like $30 K.??

This being said, it does not mean that people can't just develop Central Apnea without CHF (I am an example of that). There is also a clear scientific consensus that "UNTREATED CSA or OSA" will eventually lead to all kinds of Cardiovascular issues, including and not limited to CHF, however it is believed that the long term use of opiates can also be a driver of CSA.

As to O2 therapy for patients with CHF and Centrals, it makes a lot of sense because CSA folks (again like me) can watch their blood oxygen saturation level (SpO2) drop significantly overnight, if they are not using ASV therapy. I would imagine that supplemental Oxygen used in conjunction with PAP therapy for this group (CHF+CSA) would be very efficient and beneficial because CJHF itself is a cause for lowered SpO2.